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Home  /  FRACS Orthopaedic Surgery  /  Study notes  /  Pelvic and acetabular fractures — Young-Burgess and Letournel classification, haemodynamic control, ORIF

Pelvic and acetabular fractures — Young-Burgess and Letournel classification, haemodynamic control, ORIF

FRACS Orthopaedic Surgery LO FRACSORTHO_TRAUMA_LL_1LO FRACSORTHO_TRAUMA_LL_2 2,954 words
Free preview. This study note covers 2 learning objectives (FRACSORTHO_TRAUMA_LL_1, FRACSORTHO_TRAUMA_LL_2) from the FRACS Orthopaedic Surgery curriculum. Inside Primex you get AI-graded SAQ practice on this topic, voice viva with the AI examiner, MCQs across the full syllabus, and a curriculum tracker that ticks off every learning objective.

Overview

Pelvic ring injuries range from stable, minimally displaced fractures to life-threatening disruptions with massive haemorrhage and multisystem injury. The Young-Burgess (YB) classification is the dominant mechanistic system in trauma settings because it predicts associated injuries, transfusion requirements, and guides immediate resuscitation decisions. Mortality with non-operative management of unstable pelvic ring injuries historically approached 18%; contemporary multidisciplinary protocols incorporating binders, pelvic packing, angioembolisation, and REBOA have substantially improved survival. A single classification is insufficient for complete management decisions: the YB system (mechanism-based) and the Tile/AO-OTA system (stability-based) are complementary, with no clinically relevant difference in predictive value for mortality or transfusion requirements between the two.


Anatomy and Biomechanics

Osseoligamentous Ring

The pelvis is a true ring structure: disruption at one point mandates injury at a second point before displacement can occur. The posterior ring, sacrum, sacroiliac joints (SIJ), and posterior SIJ ligamentous complex, carries the dominant load and is the primary determinant of stability. The anterior ring (pubic symphysis, superior and inferior rami) contributes rotational stiffness.

Key Ligaments

Ligament Stability Role
Posterior SIJ complex Primary restraint to vertical displacement; strongest ligament complex in the body
Anterior SIJ ligaments Resist external rotation; disrupted in APC-II
Sacrospinous ligament Resists external rotation; disrupted in APC-II and APC-III
Sacrotuberous ligament Resists vertical and posterior shear; disrupted in APC-III and VS
Iliolumbar ligament Stabilises L5 to iliac crest
Pubic symphysis fibrocartilage Resists symphyseal diastasis

Haemorrhagic Anatomy

The majority (~85%) of pelvic haemorrhage originates from disrupted cancellous bone surfaces and the presacral/iliac venous plexuses; arterial injury accounts for ~15% but causes the most dramatic haemodynamic deterioration. The superior gluteal artery (internal iliac branch) is the most commonly injured vessel. The retroperitoneal space accommodates 3-4 litres before tamponade arrests venous bleeding; this tamponade is lost in open-book injuries because pelvic volume is increased. Closing the pelvis with a binder or external fixation restores tamponade potential.


Young-Burgess Classification

Conceptual Basis

The YB system categorises pelvic ring injuries by the direction of the causative force, expanding upon Pennal's earlier mechanistic classification. It correlates mechanism with anatomical disruption, predicts associated organ injuries, and guides haemorrhage risk stratification. Four groups are described: anteroposterior compression (APC), lateral compression (LC), vertical shear (VS), and combined mechanism (CM).

Classification

Category Subtype Anterior Injury Posterior Injury Stability Haemorrhage Risk
Lateral Compression (LC) LC-I Ipsilateral rami fracture (oblique/buckle) Sacral compression (crush) fracture on side of impact Stable Low
LC-II Rami fracture Crescent (iliac wing) fracture on side of impact Rotationally unstable Moderate
LC-III Ipsilateral LC-I or LC-II + contralateral APC injury ("windswept pelvis") Bilateral posterior disruption Rotationally unstable bilaterally High
Anteroposterior Compression (APC) APC-I Symphysis diastasis < 2.5 cm Intact SIJ; anterior and posterior SI ligaments intact Stable Low
APC-II Symphysis diastasis > 2.5 cm Anterior SI ligament disruption; posterior SI ligaments intact ("open-book") Rotationally unstable, vertically stable High
APC-III Complete symphysis disruption Complete SIJ disruption, all ligaments torn Rotationally and vertically unstable Very high
Vertical Shear (VS) VS Symphysis diastasis or rami fractures Complete SIJ disruption ± sacral fracture with vertical displacement of hemipelvis Rotationally and vertically unstable Very high
Combined Mechanism (CM) CM Variable Variable, elements of LC + VS or LC + APC Variable High-very high

Haemorrhage Correlation

Associated Injury Predictions by Pattern

Pattern Commonly Associated Injuries
APC-II/III Urethral/bladder injury, abdominal vascular injury, lumbosacral plexus injury
APC-III (equivalent open-book) More severe abdominal, spine, and extremity injuries
LC-III ("windswept") Contralateral neurovascular injury, splenic laceration
VS Lumbosacral plexus injury, sacral nerve root injury, abdominal visceral injury
CM Combination of above; highest polytrauma burden

Tile / AO-OTA Classification (Complementary System)

Type Subtype Description Stability
A A1 Avulsion / iliac wing / crest fractures, ring not involved Stable
A2 Stable, minimally displaced ring fractures Stable
B B1 Open-book (APC-II equivalent) Rotationally unstable, vertically stable
B2 LC ipsilateral (crescent fracture) Rotationally unstable, vertically stable
B3 LC contralateral / bucket-handle injury Rotationally unstable, vertically stable
C C1 Unilateral, rotationally and vertically unstable Rotationally and vertically unstable
C2 Bilateral, one side B-type, one side C-type Rotationally and vertically unstable
C3 Bilateral C-type ± acetabular fracture Rotationally and vertically unstable

Type A → non-operative; Type B → operative vs. non-operative depending on displacement and functional status; Type C → operative fixation almost always required. Type C injuries have the worst outcomes, predominantly due to associated neurological injuries.


Denis Classification of Sacral Fractures (Adjunct)

Zone Location Neurological Risk
Zone I Sacral ala (lateral to foramina) ~6% nerve injury
Zone II Foraminal region ~28% nerve injury (L5, S1 roots)
Zone III Central canal / spinal canal involvement ~57% nerve injury; bladder/bowel dysfunction

Zone III fractures are by definition the most neurologically significant. U-shaped (spinopelvic dissociation) and H-shaped sacral fractures involve bilateral Zone II/III and require lumbopelvic fixation.


Clinical Assessment

History

Primary Survey

ATLS-based. Haemodynamic instability: SBP < 90 mmHg despite initial resuscitation, or persistent HR > 120 bpm. The pelvis must be identified as the haemorrhage source by systematic exclusion of chest, abdomen, and long bone bleeding.

Examination


Investigations

Investigation Role
Pelvic AP radiograph First-line; ring disruption, diastasis, vertical shift
Inlet view AP displacement, anterior ring morphology
Outlet view Vertical displacement, sacral morphology
CT pelvis with IV contrast Defines full injury morphology, posterior ring, active arterial blush; standard of care
FAST / DPL Excludes intraperitoneal haemorrhage as alternate source
CT angiography Identifies active arterial extravasation; guides angioembolisation
Urethrogram / CT cystogram Suspected urethral/bladder injury (blood at meatus, inability to void)
MRI Not first-line; role in occult sacral fractures, ligamentous assessment, and equivocal instability in the subacute phase

Management of Haemodynamic Instability

Damage Control Resuscitation Principles

Step 1, Pelvic Binder / Sheet Wrap

Applied at the level of the greater trochanters (not the iliac crests). Reduces pelvic volume in open-book/APC injuries, promotes tamponade, allows autotransfusion from lower limbs. A simple sheet wrap is as effective as a commercial binder acutely.

Step 2, External Pelvic Fixation

Achieves ring stability and augments volume reduction. Two principal damage-control options:

Device Pin Placement Indication Limitations
Anterior external frame, iliac crest pins Between inner and outer tables of iliac wing APC-II, APC-III (temporary), LC-III Does not control posterior ring
Anterior external frame, supra-acetabular pins Supra-acetabular corridor Obese patients; better soft-tissue clearance Technical demand; proximity to lateral femoral cutaneous nerve
Pelvic C-clamp Percutaneous posterior SIJ region APC-III, VS with posterior ring instability Risk of SIJ perforation, neurovascular injury; not for comminuted sacral fractures

External fixation is a bridging/damage-control measure. Definitive posterior ring stabilisation (percutaneous iliosacral screws or ORIF) follows physiological optimisation.

Step 3, Preperitoneal Pelvic Packing (PPP)

Rationale: ~85% of pelvic haemorrhage is venous/cancellous in origin and responds to mechanical tamponade. Angioembolisation addresses arterial bleeding only; PPP targets the dominant venous source.

Technique: midline infraumbilical incision, extraperitoneal dissection into the space of Retzius and bilateral paravesical spaces, placement of 3 laparotomy packs on each side directed toward the sacrum. The peritoneum is not entered. Planned re-look with pack removal at 24-48 hours.

Evidence: institutional series demonstrate significant reduction in transfusion requirements and mortality when PPP is combined with external fixation. PPP and angioembolisation are complementary; many centres now use a combined protocol (PPP first, then angioembolisation for persistent arterial haemorrhage).

Complication of note: PPP carries a higher risk of venous thromboembolism compared with angioembolisation alone, early and aggressive VTE prophylaxis is essential once haemostasis is secured.

Step 4, Angioembolisation

Indications:

Technique: selective catheterisation of the internal iliac artery; super-selective embolisation preferred to minimise ischaemic complications (gluteal necrosis, sexual dysfunction, bladder ischaemia). Bilateral internal iliac embolisation performed when source is unlocalised.

Limitation: does not address venous/cancellous haemorrhage; availability is not universal around-the-clock.

REBOA (Resuscitative Endovascular Balloon Occlusion of the Aorta)

Mechanism: an endovascular catheter with a compliant balloon is introduced via femoral arterial access and inflated within the aorta to partially or completely occlude flow, thereby reducing distal haemorrhage, augmenting proximal (cardiac, cerebral) perfusion pressure, and bridging the patient to definitive haemorrhage control.

Aortic Zone Deployment

Zone Anatomical Location Indication
Zone I Descending thoracic aorta (T4-diaphragm) Torrential haemorrhage, near-arrest; resuscitative thoracotomy equivalent
Zone III Infra-renal aorta (above aortic bifurcation) Pelvic/junctional haemorrhage, preferred zone for isolated pelvic fracture

Zone III is the preferred deployment zone for pelvic haemorrhage because it reduces distal pelvic flow while preserving visceral and renal perfusion to a greater degree than Zone I.

Indications in pelvic fracture:

Complications:

Current evidence and limitations: systematic review data show no significant difference in health-related quality of life between REBOA and resuscitative thoracotomy. Reported complications include amputations, haematomas, and pseudoaneurysm. A clear population-wide indication for REBOA has not been established; it functions as a powerful adjunct within a multidisciplinary haemorrhage control algorithm. Dedicated training and around-the-clock endovascular capability are prerequisites.

Institutional Protocol Framework

Haemodynamically unstable pelvic fracture
 ↓
Activate MTP + TXA (within 3 hours)
 ↓
Pelvic binder at greater trochanter level
 ↓
FAST: intraperitoneal haemorrhage?
 YES → emergency laparotomy (± REBOA Zone I bridge)
 NO → continue pelvic haemorrhage algorithm
 ↓
CT angiography if haemodynamically permitting
 Arterial blush → angioembolisation
 ↓
External fixation ± REBOA Zone III if persistent instability
 ↓
Preperitoneal pelvic packing (PPP)
 ↓
Angioembolisation (if not already performed / persistent arterial bleed)
 ↓
ICU stabilisation → definitive fixation (48-72 hours)

Operative Management, Definitive Fixation

Anterior Ring

Injury Fixation Options
Symphysis diastasis (APC-II/III) Pubic symphysis plating (2- or 4-hole plate); retrograde superior ramus screws
Rami fractures (LC patterns) Antegrade or retrograde percutaneous superior ramus screws; plating for significantly displaced patterns

Posterior Ring

Injury Fixation Options
Sacral fracture Denis Zone I/II Percutaneous iliosacral (IS) screws (6.5-7.3 mm cannulated); transiliac-transsacral screws for bilateral injuries
SIJ disruption Percutaneous IS screws; open reduction + plate/screw if irreducible
Crescent fracture (LC-II) ORIF with reconstruction plate and lag screws between iliac cortical tables
U-shaped/H-shaped sacral fracture (spinopelvic dissociation) Lumbopelvic fixation (iliac screws + lumbar pedicle screws)

Timing


Complications

Complication Notes
Massive haemorrhage / exsanguination Leading cause of early mortality
Urethral / bladder injury ~10-20% of APC injuries; urethrogram before catheterisation if blood at meatus
Lumbosacral plexus / nerve root injury Most common in VS and high-grade posterior ring injuries; dominant long-term morbidity
Iatrogenic neurological injury from IS screws Pre-operative CT planning and intraoperative fluoroscopic guidance mandatory
Open fracture / perineal injury Colostomy may be required with rectal involvement; high infection risk
VTE Incidence 35-60% without prophylaxis; mechanical prophylaxis early; pharmacological once haemostasis secured; IVC filter for high-risk patients
Malunion / chronic pain Associated with residual posterior ring displacement > 1 cm
Sexual dysfunction / urinary incontinence Sacral nerve injury, urethral injury
Morel-Lavallée lesion Closed internal degloving of flank/buttock; debridement if infected; do not close over pelvic wounds without recognition
REBOA-specific: limb ischaemia Femoral access site complications; vigilance and early recognition required
PPP-specific: VTE Higher risk than angioembolisation alone

Outcomes and Prognosis


Paediatric Considerations


Key Examination Points (Viva Focus)


Sources

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Outline the Young-Burgess classification of pelvic ring injuries.
  • Anteroposterior Compression (APC): APC-I, APC-II, APC-III
  • Lateral Compression (LC): LC-I, LC-II, LC-III
  • Vertical Shear (VS)
  • Combined Mechanism (CM)
  • Based on direction of applied force and resulting instability pattern
What mechanism produces a Vertical Shear (VS) pelvic ring injury?
  • Axial load transmitted through an extended lower limb (e.g., fall from height, motor vehicle collision)
  • Results in complete disruption of anterior and posterior ring with vertical displacement of the hemipelvis
  • Most unstable pattern in the Young-Burgess system
What pubic symphysis diastasis width distinguishes APC-I from APC-II injury?
  • Threshold: 2.5 cm
  • <2.5 cm: APC-I (stable; anterior ligaments intact)
  • 2.5 cm: APC-II (anterior sacroiliac, sacrospinous, and sacrotuberous ligaments torn)

Which Young-Burgess pattern is associated with the highest transfusion requirements?
  • Vertical Shear (VS) and APC-III injuries
  • Both are rotationally and vertically unstable
  • Associated with complete posterior ring disruption and massive venous/arterial haemorrhage
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